DEĞİRMENCİ, S. Et Al. 2018. Cytosolic increased labile Zn2+ contributes to arrhythmogenic action potentials in left entricular cardiomyocytes through protein thiol oxidation and cellular ATP depletion. Journal of Trace Elements in Medicine and Biology , vol.48 , 202-212.

DEĞİRMENCİ, S., OLĞAR, Y., DURAK, A., TUNCAY, E., & TURAN, B., (2018). Cytosolic increased labile Zn2+ contributes to arrhythmogenic action potentials in left entricular cardiomyocytes through protein thiol oxidation and cellular ATP depletion. Journal of Trace Elements in Medicine and Biology , vol.48, 202-212.

DEĞİRMENCİ, SİNAN Et Al. "Cytosolic increased labile Zn2+ contributes to arrhythmogenic action potentials in left entricular cardiomyocytes through protein thiol oxidation and cellular ATP depletion," Journal of Trace Elements in Medicine and Biology , vol.48, 202-212, 2018

DEĞİRMENCİ, SİNAN Et Al. "Cytosolic increased labile Zn2+ contributes to arrhythmogenic action potentials in left entricular cardiomyocytes through protein thiol oxidation and cellular ATP depletion." Journal of Trace Elements in Medicine and Biology , vol.48, pp.202-212, 2018

DEĞİRMENCİ, S. Et Al. (2018) . "Cytosolic increased labile Zn2+ contributes to arrhythmogenic action potentials in left entricular cardiomyocytes through protein thiol oxidation and cellular ATP depletion." Journal of Trace Elements in Medicine and Biology , vol.48, pp.202-212.

@article{article, author={SİNAN DEĞİRMENCİ Et Al. }, title={Cytosolic increased labile Zn2+ contributes to arrhythmogenic action potentials in left entricular cardiomyocytes through protein thiol oxidation and cellular ATP depletion}, journal={Journal of Trace Elements in Medicine and Biology}, year=2018, pages={202-212} }