TUNCAY, E. Et Al. 2014. Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes. Oxidative Medicine and Cellular Longevity , vol.2014 .

TUNCAY, E., Okatan, E. N., Toy, A., & TURAN, B., (2014). Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes. Oxidative Medicine and Cellular Longevity , vol.2014.

TUNCAY, ERKAN Et Al. "Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes," Oxidative Medicine and Cellular Longevity , vol.2014, 2014

TUNCAY, ERKAN Et Al. "Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes." Oxidative Medicine and Cellular Longevity , vol.2014, 2014

TUNCAY, E. Et Al. (2014) . "Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes." Oxidative Medicine and Cellular Longevity , vol.2014.

@article{article, author={ERKAN TUNCAY Et Al. }, title={Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes}, journal={Oxidative Medicine and Cellular Longevity}, year=2014}