OLĞAR, Y. Et Al. 2022. Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3-adrenergic receptor signaling pathway. Journal of Cellular Physiology , vol.237, no.2 , 1353-1371.

OLĞAR, Y., DURAK, A., BİTİRİM, C. V., TUNCAY, E., & Turan, B., (2022). Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3-adrenergic receptor signaling pathway. Journal of Cellular Physiology , vol.237, no.2, 1353-1371.

OLĞAR, YUSUF Et Al. "Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3-adrenergic receptor signaling pathway," Journal of Cellular Physiology , vol.237, no.2, 1353-1371, 2022

OLĞAR, YUSUF Et Al. "Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3-adrenergic receptor signaling pathway." Journal of Cellular Physiology , vol.237, no.2, pp.1353-1371, 2022

OLĞAR, Y. Et Al. (2022) . "Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3-adrenergic receptor signaling pathway." Journal of Cellular Physiology , vol.237, no.2, pp.1353-1371.

@article{article, author={YUSUF OLĞAR Et Al. }, title={Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3-adrenergic receptor signaling pathway}, journal={Journal of Cellular Physiology}, year=2022, pages={1353-1371} }