Renal vein reconstruction for harvesting injury in kidney transplantation Kadevarik donör nefrektomi sırasında oluşan renal ven hasarında rekonstrüksiyon tekniği


Bozkurt B., ÖZKARDEŞ A. B., Tokaç M., Dumlu E. G., KILIÇ M.

Journal of Clinical and Analytical Medicine, cilt.6, sa.2, ss.256-258, 2015 (ESCI) identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 6 Sayı: 2
  • Basım Tarihi: 2015
  • Doi Numarası: 10.4328/jcam.1360
  • Dergi Adı: Journal of Clinical and Analytical Medicine
  • Derginin Tarandığı İndeksler: Emerging Sources Citation Index (ESCI), Scopus, TR DİZİN (ULAKBİM)
  • Sayfa Sayıları: ss.256-258
  • Anahtar Kelimeler: Renal Vein, Renal Transplantation, Reconstruction, VENA-CAVA, EXTENSION
  • Lokman Hekim Üniversitesi Adresli: Hayır

Özet

© 2015, Journal of Clinical and Analytical Medicine. All rights reserved.Kidney transplantation is the best treatment choice in the end-stage renal disease. In the renal transplantation, renal vein damage or shortness which occurs during cadaveric or living donor nephrectomy causes technical dif-ficulties for surgeons. The lack of the donors already especially cadaveric, the acquirement of the graft, gets very much importance. In this report, it is aimed to share the clinical experiment by which it seen, how anastomosis can become appropriate by using the renal vein which is damaged in the way that anastomosis cannot be done anyway by using cadaveric vena cava graft. The renal vein brought to length for anostomosis which is repaired by using cadaveric vena cava graft, is anastomosed successfully by becoming an end-to-side of the external iliac vein of the recipient. Vascular anastomoses are applied easily in technique. The time of the warm ischemia was under 2 hours and the kidney was functional in the post-operative period. Renal vein trom-bosis was not observed. The renal vein damage occured during cadaveric or living donor nephrectomy, can be repaired by some methods. In the kidneys in which vein requirement is done, the success rates are rather high although acute tubular necrosis and delayed function can be seen more.