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HAN S., BAL N. B., SADİ G., USANMAZ S. E., Tuglu M. M., ULUDAĞ M. O., ...More
VASCULAR PHARMACOLOGY, vol.113, pp.38-46, 2019 (SCI-Expanded)
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Publication Type:
Article / Article
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Volume:
113
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Publication Date:
2019
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Doi Number:
10.1016/j.vph.2018.11.004
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Journal Name:
VASCULAR PHARMACOLOGY
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Journal Indexes:
Science Citation Index Expanded (SCI-EXPANDED), Scopus
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Page Numbers:
pp.38-46
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Keywords:
Hypertension, Endoplasmic reticulum stress, Vascular dysfunction, TUDCA, GROWTH-FACTOR RECEPTOR, SMOOTH-MUSCLE-CELLS, ENDOTHELIAL DYSFUNCTION, OXIDATIVE STRESS, ANTIOXIDANT STATUS, TYROSINE KINASE, CARDIAC DAMAGE, BLOOD-PRESSURE, ER STRESS, RESISTANCE
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Lokman Hekim University Affiliated:
No
Abstract
Hypertension has complex vascular pathogenesis and therefore the molecular etiology remains poorly elucidated. Endoplasmic reticulum stress (ERS), which is a condition of the unfolded/misfolded protein accumulation in the endoplasmic reticulum, has been defined as a potential target for cardiovascular disease. In the present study, the effects of ERS inhibition on hypertension-induced alterations in the vessels were investigated.